Vegetarian Miso Soup With 6 Minute Eggs
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Her research actions include a health status monitoring program in seals and cetaceans from the German North and Baltic Sea and Black Sea cetaceans with special emphasis on morbillivirus infections. Müller G, Siebert U, vapebeased Wünschmann A, Baumgärtner W. Immunohistological and serological investigation of morbillivirus infection in harbour porpoises (Phocoena phocoena) from the Baltic and North Sea. Frisk AL, König M, Moritz A, Baumgärtner W.
Detection of canine distemper virus nucleoprotein RNA by reverse transcription-PCR utilizing serum, entire blood, and cerebrospinal fluid from canine with distemper. Kennedy S, Kuiken T, Jepson PD, Deaville R, Forsyth M, Barrett T, Mass die-off of Caspian seals brought on by canine distemper virus. Antibodies to canine distemper virus in Antarctic seals. Pathological findings indicative of distemper in European seals.
Kennedy S. A overview of the 1988 European seal epizootic. Up to now, migrating Arctic seal species, such as harp seals from Greenland, have been suspected as carriers that introduced a morbillivirus into an immunologically naive inhabitants (15). This species could have served as a reservoir that maintains the circulation of PDV. Reijnders PJH, Brasseur S, Abt KF, Siebert U, Stede M, Tougaard S. The harbour seal population in the Wadden Sea as revealed by the aerial surveys in 2003.
Wadden Sea Newsletter. The RNA sequences of the current virus isolates showed a virus inhabitants along the German coast during this epidemic that was almost an identical to the isolates from the Netherlands and Denmark in 2002 and that had a excessive id to the isolate from 1988 (6). Protective morbillivirus-specific antibody titers had been detectable in just a few seals from German waters before the outbreak in 2002, suggesting a high susceptibility for morbillivirus infection in this naive inhabitants.
As well as, genetic number of a less prone population originating from the survivors of the 1988 outbreak might have resulted in a decrease variety of deaths during the second epidemic. Several epizootics of infectious diseases in marine mammals with increases in air temperature have been observed, indicating that environmental influences might have also resulted within the emergence of latest epidemics (16).
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Her research actions include a health status monitoring program in seals and cetaceans from the German North and Baltic Sea and Black Sea cetaceans with special emphasis on morbillivirus infections. Müller G, Siebert U, vapebeased Wünschmann A, Baumgärtner W. Immunohistological and serological investigation of morbillivirus infection in harbour porpoises (Phocoena phocoena) from the Baltic and North Sea. Frisk AL, König M, Moritz A, Baumgärtner W.
Detection of canine distemper virus nucleoprotein RNA by reverse transcription-PCR utilizing serum, entire blood, and cerebrospinal fluid from canine with distemper. Kennedy S, Kuiken T, Jepson PD, Deaville R, Forsyth M, Barrett T, Mass die-off of Caspian seals brought on by canine distemper virus. Antibodies to canine distemper virus in Antarctic seals. Pathological findings indicative of distemper in European seals.
Kennedy S. A overview of the 1988 European seal epizootic. Up to now, migrating Arctic seal species, such as harp seals from Greenland, have been suspected as carriers that introduced a morbillivirus into an immunologically naive inhabitants (15). This species could have served as a reservoir that maintains the circulation of PDV. Reijnders PJH, Brasseur S, Abt KF, Siebert U, Stede M, Tougaard S. The harbour seal population in the Wadden Sea as revealed by the aerial surveys in 2003.
Wadden Sea Newsletter. The RNA sequences of the current virus isolates showed a virus inhabitants along the German coast during this epidemic that was almost an identical to the isolates from the Netherlands and Denmark in 2002 and that had a excessive id to the isolate from 1988 (6). Protective morbillivirus-specific antibody titers had been detectable in just a few seals from German waters before the outbreak in 2002, suggesting a high susceptibility for morbillivirus infection in this naive inhabitants.
As well as, genetic number of a less prone population originating from the survivors of the 1988 outbreak might have resulted in a decrease variety of deaths during the second epidemic. Several epizootics of infectious diseases in marine mammals with increases in air temperature have been observed, indicating that environmental influences might have also resulted within the emergence of latest epidemics (16).
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