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Her analysis actions embody a well being standing monitoring program in seals and cetaceans from the German North and Baltic Sea and Black Sea cetaceans with particular emphasis on morbillivirus infections. Müller G, Siebert U, Wünschmann A, Baumgärtner W. Immunohistological and Premium eLiquid serological investigation of morbillivirus infection in harbour porpoises (Phocoena phocoena) from the Baltic and North Sea. Frisk AL, König M, Moritz A, Baumgärtner W.
Detection of canine distemper virus nucleoprotein RNA by reverse transcription-PCR utilizing serum, entire blood, and cerebrospinal fluid from canines with distemper. Kennedy S, Kuiken T, Jepson PD, Deaville R, Forsyth M, Barrett T, Mass die-off of Caspian seals attributable to canine distemper virus. Antibodies to canine distemper virus in Antarctic seals. Pathological findings indicative of distemper in European seals.
Kennedy S. A review of the 1988 European seal epizootic. Previously, migrating Arctic seal species, such as harp seals from Greenland, have been suspected as carriers that launched a morbillivirus into an immunologically naive population (15). This species might have served as a reservoir that maintains the circulation of PDV. Reijnders PJH, Brasseur S, Abt KF, Siebert U, Stede M, Tougaard S. The harbour seal inhabitants within the Wadden Sea as revealed by the aerial surveys in 2003.
Wadden Sea Newsletter. The RNA sequences of the current virus isolates confirmed a virus population along the German coast throughout this epidemic that was virtually equivalent to the isolates from the Netherlands and Denmark in 2002 and that had a high id to the isolate from 1988 (6). Protective morbillivirus-particular antibody titers were detectable in just a few seals from German waters before the outbreak in 2002, suggesting a high susceptibility for morbillivirus infection in this naive inhabitants.
In addition, genetic collection of a less vulnerable population originating from the survivors of the 1988 outbreak may need resulted in a lower variety of deaths throughout the second epidemic. Several epizootics of infectious diseases in marine mammals with will increase in air temperature have been noticed, indicating that environmental influences may have also resulted within the emergence of new epidemics (16).
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Her analysis actions embody a well being standing monitoring program in seals and cetaceans from the German North and Baltic Sea and Black Sea cetaceans with particular emphasis on morbillivirus infections. Müller G, Siebert U, Wünschmann A, Baumgärtner W. Immunohistological and Premium eLiquid serological investigation of morbillivirus infection in harbour porpoises (Phocoena phocoena) from the Baltic and North Sea. Frisk AL, König M, Moritz A, Baumgärtner W.
Detection of canine distemper virus nucleoprotein RNA by reverse transcription-PCR utilizing serum, entire blood, and cerebrospinal fluid from canines with distemper. Kennedy S, Kuiken T, Jepson PD, Deaville R, Forsyth M, Barrett T, Mass die-off of Caspian seals attributable to canine distemper virus. Antibodies to canine distemper virus in Antarctic seals. Pathological findings indicative of distemper in European seals.
Kennedy S. A review of the 1988 European seal epizootic. Previously, migrating Arctic seal species, such as harp seals from Greenland, have been suspected as carriers that launched a morbillivirus into an immunologically naive population (15). This species might have served as a reservoir that maintains the circulation of PDV. Reijnders PJH, Brasseur S, Abt KF, Siebert U, Stede M, Tougaard S. The harbour seal inhabitants within the Wadden Sea as revealed by the aerial surveys in 2003.
Wadden Sea Newsletter. The RNA sequences of the current virus isolates confirmed a virus population along the German coast throughout this epidemic that was virtually equivalent to the isolates from the Netherlands and Denmark in 2002 and that had a high id to the isolate from 1988 (6). Protective morbillivirus-particular antibody titers were detectable in just a few seals from German waters before the outbreak in 2002, suggesting a high susceptibility for morbillivirus infection in this naive inhabitants.
In addition, genetic collection of a less vulnerable population originating from the survivors of the 1988 outbreak may need resulted in a lower variety of deaths throughout the second epidemic. Several epizootics of infectious diseases in marine mammals with will increase in air temperature have been noticed, indicating that environmental influences may have also resulted within the emergence of new epidemics (16).
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